Meanings of esophageal varices without mention of bleeding in English

Fifteen variables were analysed to identify the presence of large esophageal varices.

One hundred and fifteen patients with cirrhosis and esophageal varices were studied.

Every third patient with parenchymal liver disease bleeds from esophageal varices.

Sclerotherapy was repeated to eradicate esophageal varices and follow-up endoscopic examination were performed.

Endoscopy showed large esophageal varices that were treated with band ligation.

Multivariate analysis was performed to identify the markers best correlated with oesophageal varices.

The principal treatment for bleeding oesophageal varices is endoscopic ligation.

The diagnostic accuracy for medium to large oesophageal varices using these three factors was 86%.

We were unable to find any factors differentiating bleeders from non-bleeders, although these results do not justify prophylactic surgery for oesophageal varices.

The conclusion was drawn that the results of endoscopic sclerotherapy of bleeding oesophageal varices was effective and clearly related to liver function.

Portal vein invasion is a risk factor for subsequent variceal bleed.

Thirty-eight patients died during follow-up, and 16 had a variceal bleed.

Patients with poor hepatic function are at higher risk of re-bleeding and death after acute gastric variceal bleed.

The four rebleeding episodes consisted of an esophageal variceal bleed, a gastric variceal bleed, a duodenal ulcer bleed, and a bleed caused by hemorrhagic gastritis.

Recurrent variceal bleeding is common and is associated with a high mortality.

Shunt therapy, preferably the placement of a TIPS, is indicated for refractory acute variceal bleeding.

Of 55 children with acute variceal bleeding, 39 had band ligation and 16 had injection sclerotherapy.

Its efficacy for the treatment of bleeding esophageal varices is less certain, and more controlled studies are necessary.

Management of variceal hemorrhage includes emergency treatment of bleeding esophageal varices and prophylactic treatment for the prevention of first bleeding or rebleeding.

Fifty-three patients with bleeding esophageal varices received a non-shunting operation-esophagealtransection and devascularization of the proximal stomach with splenectomy-fromMarch 1977 to October 1986.

Bleeding esophageal varices, spontaneous bacterial peritonitis and hepatic encephalopathy are major complications of cirrhosis and traditional indications for liver transplantation evaluation.

The principal treatment for bleeding oesophageal varices is endoscopic ligation.

The conclusion was drawn that the results of endoscopic sclerotherapy of bleeding oesophageal varices was effective and clearly related to liver function.

Conclusions: Emergency injection of acrylate glue is safe and effective for the treatment of acute bleeding oesophageal varices and does not hamper subsequent variceal ligation.